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On November 5, the internationally renowned academic journal Nature Genetics published online a collaborative research achievement between the Oil Crops Research Institute and Huazhong Agricultural University. The research reveals the patterns and mechanisms of how structural variations in the rapeseed genome regulate gene expression and phenotypic diversity.

In this study, the authors compared the alternative splicing (AS) landscapes of a susceptible parental line and two mutagenized B. napus lines with contrasting levels of black rot resistance. Different types of AS events were identified in these B.

This study assembled a high-quality chromosome-scale genome of a B. napus mutant dc1 with a dwarf and compact plant architecture. With the genome as a method of fine mapping and gene cloning deal with widespread structure variation, we identified a “master” gene BnaA04.

This study identified a natural variation of RLK902 conferring resistance to SRD and GMD in a natural population of B. napus. Importantly, knocking out RLK902 by genome editing displayed a higher level of disease resistance in B.

Abstract Clubroot disease caused by the soil-borne Plasmodiophora brassicae is devastating to Brassicaceae crops and spreading rapidly in China in recent years, resulting in great yield losses annually. Virulence of P.

BnaOmics is a comprehensive central platform for genomics, transcriptomics, epigenomics, and variations in Brassica napus (B. napus). It integrates 26 B. napus reference genomes, re-sequencing data of 2,885 accessions, 8,736,523 high-quality SVs, 112,886 high-quality presence/absence variants (PAVs), thousands of transcriptomes, and hundreds of epigenomes.

On March 14th at 11 AM local time (6 PM Beijing time), the thesis defense of doctoral student Li Yang from the joint training program of CAAS Graduate School/Oil Crops Research Institute and Wageningen University was held at Wageningen University in the Netherlands.

Highlights A novel flowering time locus BnaA02.Ebm, covering 905 kb region, was detected on chromosome A02 by BSA-seq and fine mapping. Combination of the expression and variation analyses, regional association and haplotype analyses, and transcriptome analysis were helpful to identify the potential candidate gene, cytokinin response factor 10 (CRF10), and mine the favorable allelic variations.